LSAT 120 – Section 4 – Question 10

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Question
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Type Tags Answer
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Curve Question
Difficulty
Psg/Game/S
Difficulty
Explanation
PT120 S4 Q10
+LR
+Exp
Flaw or descriptive weakening +Flaw
Causal Reasoning +CausR
Link Assumption +LinkA
A
1%
152
B
93%
162
C
4%
156
D
0%
150
E
1%
152
124
134
144
+Easiest 146.628 +SubsectionMedium


J.Y.’s explanation

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Researcher: All defects in short-term memory are caused by a malfunction of a part of the brain called the hippocampus. In short-term memory, the mind holds a piece of information for only a few moments, after which it is either stored in long-term memory or forgotten. Learning is the accumulation of new information in long-term memory. Thus, whenever a child exhibits a learning deficit, the hippocampus is malfunctioning.

Summarize Argument: Phenomenon-Hypothesis
The researcher concludes that whenever a child exhibits a learning deficit, the hippocampus is malfunctioning. Why? Because, if memory fails to work correctly, it leads to a learning deficit. And all short-term memory failures are caused by hippocampus malfunctions.

Identify and Describe Flaw
This is the cookie-cutter flaw of confusing sufficiency and necessity. The author implicitly argues that hippocampus memory malfunctions always lead to learning deficits. Therefore, learning deficits are always the result of hippocampus malfunctions.
The problem is that we don’t know that—there could be learning deficits that are the result of factors other than hippocampus malfunctions.

A
draws a general conclusion based on too small a sample of learning deficits
We have no indication that the researcher’s sample size of learning deficits is insufficient.
B
presumes, without giving justification, that all learning deficits in children involve short-term memory
The author has to presume this: if he doesn’t, there could be learning deficits not caused by memory problems, and therefore not necessarily caused by hippocampus malfunctions.
C
presumes, without giving justification, that short-term memory is disabled whenever the hippocampus is disabled
This is the reverse of what the author presumes. Namely, he thinks that the hippocampus is disabled whenever short-term memory is disabled.
D
fails to quantify precisely the length of time during which the mind holds a piece of information in short-term memory
The precise length of time is irrelevant; all the author needs to establish for his argument is that the length of time is limited.
E
takes for granted that learning deficits in adults have a cause unrelated to the cause of learning deficits in children
The author doesn’t presume this—learning deficits in adults aren’t mentioned here—so it can’t be the flaw.

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